Journal of Regenerative Medicine ISSN: 2325-9620

Giacomo Lanzoni

Giacomo Lanzoni
Diabetes Research Institute, University of Miami, USA

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Biography

Giacomo Lanzoni earned his PhD in 2010 at the University of Bologna (Italy) with his work on stem cells from adult and placental tissues. He received the “Proni-Caravita” award to support 3 years of postdoctoral studies abroad. He conducted his postdoctoral studies at the Diabetes Research Institute (DRI, University of Miami, USA) with Dr Luca Inverardi. Dr Lanzoni is now Assistant Scientist at the DRI. His research is focused on the pathogenesis of Type 1 Diabetes in humans and on regenerative approaches for this disease. He is investigating the activation of pathways for islet regeneration in the period that precedes the clinical onset of Type 1 Diabetes. He recently described the activation of tolerogenic pathways in human pancreatic islets at the initial stages of the disease: intriguingly, these pathways could disable the innate immune system and may represent mechanisms of viral hiding in islets. He is studying stem cells of the adult pancreas (biliary tree stem cells) that have the potential to mature into insulin producing beta cells. He was recently awarded the “Marc S. Goodman Oustanding Young Scientist” prize for these studies. Dr Lanzoni has been involved in stem cell research for 10 years. He contributed to the development of stem cell-based approaches for inflammatory bowel disease and diabetes. He was adjunct Assistant Professor in Histology and Embryology, Lecturer in Stem Cells and Cell Therapy at the University of Bologna. Through the years, he supervised the work of 12 students in their BSc, MSc, PhD and MD theses. Dr Lanzoni has developed skills in research designing, scientific writing and presentation delivering. He is author of 18 publications in international journals and 3 book chapters.

Research Interest

His research is focused on the pathogenesis of Type 1 Diabetes in humans and on regenerative approaches for this disease. He is investigating the activation of pathways for islet regeneration in the period that precedes the clinical onset of Type 1 Diabetes.

Publications

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