Journal of Athletic EnhancementISSN: 2324-9080

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Research Article, J Athl Enhancement Vol: 4 Issue: 2

Live High-Train Low Altitude Training: Responders and Non-Responders

Hamlin MJ1*, Manimmanakorn A1,2, Creasy RH3 and Manimmanakorn N1,4
1Department of Tourism, Sport and Society, Lincoln University, New Zealand
1Department of Physiology, Faculty of Medicine, Khon Kaen University, Khon Kaen 40002, Thailand
1High Performance Sports Physiologist, Triathlon, New Zealand
4Department of Rehabilitation, Faculty of Medicine, Khon Kaen University, Khon Kaen 40002, Thailand
Corresponding author : Michael John Hamlin
Department of Tourism, Sport and Society, Lincoln University, Christchurch 7647, New Zealand
Tel: +64-3-423-0489; Fax: +64-3-325-3857
E-mail: mike.hamlin@lincoln.ac.nz
Received: February 24, 2015 Accepted: May 20, 2015 Published: May 26, 2015
Citation: Hamlin MJ, Manimmanakorn A, Creasy RH, Manimmanakorn N (2015) Live High-Train Low Altitude Training: Responders and Non-Responders. J Athl Enhancement 4:1. doi:10.4172/2324-9080.1000193

Abstract

Live High-Train Low Altitude Training: Responders and Non-Responders

Objective: Investigate differences between athletes that responded (improved performance) compared to those that did not, after a 20- day “live high-train low” (LHTL) altitude training camp. Methods: Ten elite triathletes completed 20 days of live high (1545-1650 m), train low (300 m) training. The athletes underwent (i), two 800-m swimming time trials at sea-level (1 week prior to and 1 week after the altitude camp) and (ii) two 10-min standardised submaximal cycling tests at altitude on day 1 and day 20 of the altitude camp. Acute mountain sickness (AMS) was also measured during the camp. Based on their 800-m swimming time trial performances, athletes were divided into responders (improved by 3.2 ± 2.2%, mean ± SD, n=6) and non-responders (decreased by 1.8 ± 1.2%, n=4). Results: Compared to non-responders, the responders had lower exercise heart rates (-6.3 ± 7.8%, mean ± 90% CL, and higher oxygen saturations (1.2 ± 1.3%) at the end of the 10-min submaximal test after the camp. Compared to the responders, the non-responders had substantially higher VE and VE/VO2 during the submaximal test on day 1 of the altitude training camp, and a substantially higher RER during the submaximal test on day 20 of the camp. As a result of the altitude training, exercise economy of the non-responders compared to the responders deteriorated (i.e., non-responders required more oxygen per watt). Non-responders were 3.0 times (90% CL=0.5-16.6) more likely to suffer symptoms of acute mountain sickness during first 5 days of altitude compared to responders. Conclusion: Changes in SpO2, heart rate and some respiratory variables during exercise and resting AMS scores may help determine athletes that respond to LHTL altitude training camps from athletes that fail to respond to such training.

Keywords: Oxygen saturation; Acute mountain sickness; Submaximal exercise; Altitude training; Triathlon; Elite athletes

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